Abstract
Itch pathogenesis involves modulation of the neuroimmune axis, with several immunosuppressants such as azathioprine1 and mycophenolate mofetil2 demonstrating significant anti-pruritic activity in subsets of patients with pruritus. These immunosuppressive agents are contraindicated in patients with malignancy, and thus there is a need for novel anti-pruritic agents without significant immunosuppressive effects in neoplastic conditions such as cutaneous T cell lymphoma (CTCL).
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